Download A Case-Based Guide to Clinical Endocrinology by Steven Jon Russell, Karen Klahr Miller (auth.), Terry F. PDF

By Steven Jon Russell, Karen Klahr Miller (auth.), Terry F. Davies (eds.)

Written circumstances offer a considerate evaluate of recent administration and contemplation of the literature, even more so than even a case presentation with no the sufferer. hence, in recent times the written or ready case has develop into definitely the right discussion board for demonstrating the right way to deal with a scientific challenge to the best good thing about the sufferer. In A Case-Based consultant to medical Endocrinology, individual clinicians supply stimulating guide and insights right into a wide array of endocrine matters, educating readers glossy administration of the stipulations defined and supplying exact access into the literature. Importantly, a few of the vital parts within the present curriculum for scientific endocrine education as urged via the organization of software administrators in medical Endocrinology, Diabetes and Metabolism are lined during this textual content. to augment studying and retention of information, every one writer has additionally supplied a number of selection questions following the case experiences. scholars, citizens, fellows, and practitioners will locate those examples to be an effective method to evaluate their knowing and elevate their abilities and skills in sufferer research and administration.

In all, A Case-Based consultant to medical Endocrinology bargains a finished quantity of concise case-studies that would end up to be of important value to all clinicians practising during this box.

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Extra resources for A Case-Based Guide to Clinical Endocrinology

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Causing visual symptoms optic chiasm (CN II) compression 1 Pituitary Apoplexy 17 C. Causing ophthalmoplegia CN III or VI involvement D. Causing diplopia cranial nerve IV involvement Answer: C. Headache is the most common and nearly universal symptom in apoplexy, but in most cases it is not thought be associated with involvement of CN V. Ophthalmoplegia in general is more common than visual impairment, and cranial nerves III and VI are most commonly affected. CN VI and IV impairment may not be obvious with casual observation, so formal examination of extraocular movements is important.

1% (normal, 20–25%). Background Amiodarone is a class III antiarrhythmic agent that has been associated with a wide array of serious cardiac, pulmonary, hepatic, ocular, and thyroid side effects. 5% of the mass of amiodarone is made up of iodine, a patient taking a daily dose of 200 mg of amiodarone consumes about 75 mg of organic iodine daily [1]. Metabolism of this amount of iodine releases an amount of free iodine into the circulation that is about 20 to 40 times higher than the daily iodine intake in the United States [1].

A Case-Based Guide to Clinical Endocrinology, C Humana Press, Totowa, NJ 2008 41 42 P. S. Cooper movements. The thyroid was enlarged approximately twofold, and it was firm and nontender. There were no discrete nodules appreciated and no bruits auscultated. Lungs, heart, and abdomen were unremarkable. The skin was warm and moist, there was no tremor, and deep tendon reflexes were normal. Antithyroperoxidase (anti-TPO) and antithyroglobulin antibody titers were low, and thyroid-stimulating immunoglobulins (TSIs) activity was normal at 92% (normal, <125%).

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