By Ian Stuart-Hamilton
With the world's inhabitants getting more and more older, there hasn't ever been a extra urgent want for the research of previous age and growing old. An creation to Gerontology presents a wide-ranging creation to this crucial subject. by means of assuming no earlier specialist wisdom and heading off jargon, this ebook will consultant scholars via the entire major topics in gerontology, overlaying either conventional parts, corresponding to organic and social growing older, in addition to extra modern parts, akin to expertise, the humanities, sexuality and schooling of older adults. An advent to Gerontology is written by way of a crew of overseas authors with multidisciplinary backgrounds who draw proof from a number of various views and traditions.
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Extra resources for An Introduction to Gerontology
One of the most pervasive public misconceptions about biogerontology is the idea that the goal of anti-ageing medicine is to make old people live longer by merely extending life and consequently extending age-related debilitation and suffering. This is known as the Tithonus error. In Greek mythology, Tithonus was a mortal to whom Zeus conceded immortality but not eternal youth, rendering Tithonus increasingly debilitated and demented as he aged. Contrary to the immortality granted to Tithonus, the goal of biogerontology is to extend healthy lifespan by postponing disease and extending the healthy period of life.
The first of these genes was identified somewhat serendipitously by Andrzej Bartke and colleagues. While working on a mouse strain called the Ames dwarf mouse, which has a small body size due to a lack of growth hormone, Bartke and colleagues noticed that animals were living longer than expected. Intrigued, they conducted a careful study in which they showed that indeed the Ames dwarf mice can live roughly 50 per cent longer than controls (Brown-Borg, Borg, Meliska and Bartke, 1996). Ames dwarf mice are the result of a mutation in a single gene called Prop1, which is essential for the production of growth hormone and prolactin in the pituitary gland.
Of even greater interest, a handful of genes have been shown to increase lifespan in mice, most of which are related to the mammalian equivalent of the insulin/IGF1 pathway. The first of these genes was identified somewhat serendipitously by Andrzej Bartke and colleagues. While working on a mouse strain called the Ames dwarf mouse, which has a small body size due to a lack of growth hormone, Bartke and colleagues noticed that animals were living longer than expected. Intrigued, they conducted a careful study in which they showed that indeed the Ames dwarf mice can live roughly 50 per cent longer than controls (Brown-Borg, Borg, Meliska and Bartke, 1996).