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By J. Esparza-Gordillo

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The role of the hairless (hr) gene in the regulation of hair follicle catagen transformation. 1, (July 1999), pp. J. W. (2011) Topical application of two condensed tannins from the root of Rosa multiflora Thunberg for the treatment of atopic dermatitis (AD) in NC/Nga Mice. , Bevan, S. & Patapoutian, A. (2002). A heat-sensitive TRP channel expressed in keratinocytes. 5575, (June 2002), pp. L. & Kusakabe M. (2002). Hague (Hag). A new mouse hair mutation with an unstable semidominant allele. 2, (October 2002), pp.

E. (2003). TRP channels as cellular sensors. 6966, (December 2003), pp. A. C. (2010). Occupational mouse allergen exposure among non-mouse handlers. 12, (December 2010), pp. , Amagai, M. H. (2009). A homozygous frameshift mutation in the mouse Flg gene facilitates enhanced percutaneous allergen priming. 5, (April 2009) pp. W. (1996). F. Lyon, S. M. ), pp. S. (2003). Allergy and hypersensitivity. Nature versus nurture in allergy and hypersensitivity. 6, (December 2003), pp. M. (1982). Atopic dermatitis.

Mouse Models for Atopic Dermatitis Developed in Japan 29 The Nh mutation is controlled by a single dominant mutation that occurred in the transient receptor potential (TRP) cation channel, subfamily V member 3 (Trpv3). , 2007) because they are critical mediators in sensory systems and respond to temperature, touch, pain and other important stimuli. TRP channels are divided into six main subfamilies, including TRPV (Clapham, 2003). , 2002). , 2007). By studying dermatitis in DS-Nh mice, two major pathways have been identified; one is the interaction between the gain-of-function Trpv3 mutation and NKT cells with the T-cell receptor Vand the other is the synergistic production of interleukin-13 (IL-13) through the activation of Toll-like receptor 2 by staphylococcal enterotoxin C-producing S.

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